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Diabetes is a disorder that affects the way the body handles digested carbohydrates in a way that results in excessively high levels of blood glucose.

Blood sugar levels are regulated by insulin, a hormone secreted by the pancreas, which releases it in response to carbohydrate consumption. Insulin causes the cells of the body to absorb glucose from the blood, where it then serves as fuel for cellular functions. In diabetics, cells either become unresponsive to insulin, or insufficient insulin is produced by the pancreas. There is a complex interaction between other hormones that can also affect the insulin response.

If early symptoms are neglected, diabetes can cause extremely severe health complications, including blindness, circulatory failure, peripheral neuropathy (degeneration of sensing nerves) and kidney failure. Around eight percent of Western populations have symptoms of diabetes. This means that around sixteen million people in the US alone have been diagnosed with the disease. The American Diabetes Association (ADA) estimates that in the US, diabetes accounts for 178,000 deaths, as well as 54,000 amputations, and 12,000-24,000 cases of blindness annually. Blindness is twenty-five times even more common among diabetic patients in comparison with nondiabetics, and lifespan is on average 10 years under the norm. Complications of diabetes exceed both heart disease and cancer as the major cause of death in America.

Traditional diagnostic standards for diabetes have been fasting plasma glucose levels greater than 140 mg/dL on 2 occasions and plasma glucose greater than 200 mg/dL following a 75-gram glucose load. However, the ADA recently reduced the criteria for a diabetes diagnosis to fasting plasma glucose levels equal to or higher than 126 mg/dL. Fasting plasma levels outside the normal limit indicate a need for further testing, usually by repeating the fasting plasma glucose check and (if indicated) initiating an oral glucose tolerance test.

The many symptoms of diabetes include excessive urination, excessive thirst and hunger, sudden weight loss, blurred vision, delay in healing of wounds, dry and itchy skin, repeated infections, fatigue and headache. While suggestive of diabetes, these symptoms can also be caused by other factors, and therefore anyone with symptoms suspicious of the disease should be tested.

There are 2 different varieties of diabetes.

Type I Diabetes (juvenile diabetes, also known as insulin-dependent diabetes)

About 5-10% of cases of diabetes are Type I. The cause of Type I diabetes is failure of the pancreas to make insulin. In very rare cases this is as a result of a genetic mutation, but much more commonly, the pancreatic cells which secrete the hormone are destroyed by the individual's own immune system. Such autoimmune responses are increasingly common, and may be triggered by environmental factors, including certain viral infections. 

Type I diabetes is most prevelant in the caucasian population and has a hereditary component. If untreated, Type I or juvenile diabetes can lead to death within two to three months of the onset, as the cells of the body starve because they no longer receive the hormonal prompt to absorb glucose. While a great majority of Type I diabetics are young (hence the term Juvenile Diabetes), the condition can develop at any age. Autoimmune diabetes is diagnosed by an immunological assay which shows the presence of anti-insulin/anti-islet-cell antibodies.

Type II Diabetes (non insulin dependent diabetes, also known as adult onset diabetes)

This type of diabetes is a consequence of body cells becoming resistant to the effects of insulin. It accounts for 90-95% of cases, and tends to develop slowly in adult life through symtomatic 'pre-diabetic' stages. In many cases the pancreas is producing an adequate amount of insulin, but the cells of the body have become unresponsive to its effect due to the chronically high level of the hormone. Finally the pancreas will exhaust its over-active secretion of the hormone, and insulin levels then fall to beneath normal and tend to remain there.

A tendency towards Type II diabetes is hereditary, although it is unlikely to develop in normal-weight individuals eating a low- or even moderate-carbohydrate diet. Obese, sedentary individuals who eat poor-quality diets containing excessive amounts of refined starch or sugar, which constantly activates pancreatic insulin secretion, are prone to develop insulin resistance.

Non-Eupopeans, whose traditional diets never included refined starch or sugar until these items were introduced by Europeans, often have very high rates of diabetes -up to five times the rate of caucasians, whose ancestors have consumed large quantities of refined starches for many generations.  Though Type II diabetes isn't as immediately disastrous as Type I, it can lead to health complications after many years and cause serious disability and shortened lifespan if no action is taken to control it. As with Type I diabetes, the condition develops primarily in a certain age group, in this case patients over forty (which is why it's typically termed Adult Onset Diabetes); however, with the rise in childhood and teenage obesity, this condition is being seen for the first time in school children as well.

blood testing

If treatment is neglected, both Type I and Type II diabetes can lead to life-threatening complications such as kidney damage (nephropathy), heart disease, nerve damage (neuropathy), retinal damage and blindness (retinopathy), and hypoglycemia (drastic reduction in glucose levels). Diabetes damages blood vessels, especially smaller end-arteries, leading to very severe and premature atherosclerosis. Diabetics are prone to foot problems because neuropathy, which afflicts about ten percent of patients, causes their feet to lose sensation. Foot injuries, common in day-to-day living, go unnoticed, and these injuries cannot heal because of atherosclerotic blockage of the microscopic arteries in the foot. Gangrene and subsequent amputation of toes, feet or even legs is the result for many elderly patients with poorly-controlled diabetes. Usually these sequelae are seen sooner in Type I than Type II diabetes, because Type II patients have a small amount of their own insulin production left to buffer changes in blood sugar levels.

Type I diabetes is a severe disease and there is no known permanent cure for it. Nonetheless, the symptoms can be controlled by strict dietary monitering and insulin injections. Implanted pumps which release insulin immediately in response to changes in blood glucose are becoming available.

Type II diabetes however is preventable and if detected, manageable or even curable by dietary changes alone. But as so often happens, clinical theory is defeated by human nature in this case, as many diabetics (and many obese people without diabetes) find it personally impossible to lose weight or even stick to a diet free of starchy, sugary junk food. Type II diabetes is therefore frequently treated with drugs which restore the body's response to its own insulin, and in a few cases injections of insulin.

See Salacia oblangata in the Supplements - Herbal section, and further information in the articles (hover your mouse cursor over [Diabetes] in the menu on the left of this page.)

The fact is however that diabetes management through drugs is now a $245 billion industry, and so no effort is made by the pharmaceutical industry to find a permanent cure for Type II diabetes. This has been left to independent researchers who, unable to publish in any industry sponsored journals, now offer practical, workable protocols for eliminating diabetes or pre-diabetes commercially.

Please click the link below to watch a video presentation for one of the best of these, now used by over 41,000 people to eliminate injections and food watching.

Click Here for the Video

(The video presentation will open in a new tab or window)




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